Fig. 1From: LncRNAs H19 and HULC, activated by oxidative stress, promote cell migration and invasion in cholangiocarcinoma through a ceRNA mannerAnalysis of Inflammation-associated lncRNAs. a Heat maps of differential lncRNA profiles are based on studies upon lncRNAs responded to hypoxic and inflammatory factor. Each of the GEO data has suggested the aberrantly lncRNA related with inflammation and/or oxidative stress. For example, GSE76743 identified endothelial cell response to hypoxia. Others: GSE57539 (PBS_VS_IL6 treated MDA-MB-231 cells), GSE67106 (Non_ VS_ inflamed Crohn’s disease), GSE55146 (Non_ VS_ cystic fibrosis bronchial epithelium), and GSE70544 (Normoxia_vs_Hypoxia in proximal tubular epithelial cells (PTECs)). b LncRNAs stimulated by short-term oxidative stress using H2O2 in QBC939, SK-cha-1, and RBE cells. c LncRNAs stimulated by long-term oxidative stress using glucose oxidase in QBC939, SK-cha-1, and RBE cellsBack to article page